Non Insulin-Dependent Diabetes

B. Potential effects of Type II diabetes mellitus.

C. Material to be covered in this examination.

B. Suggestion for future exploration.

C. Importance of material reviewed.

DIABETES MELLITUS TYPE II: NURSING ASSESSMENT AND MANAGEMENT

Diabetes is a disease that develops as a result of the body's inability to make appropriate use of food consumed, which in turn is the result of an insufficiency of insulin (Robertson, 1995, p. 35). Type I diabetes is defined as insulin-dependent diabetes, while Type II diabetes is defined as non insulin-dependent diabetes. A wide variety of complications may result from this chronic disease (Betts, Betts, & Betts, 1995, p. 415). This research examines Type II diabetes mellitus from a nursing perspective with emphasis on assessment, diagnosis, inte

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not known whether insulin resistance causes NIDDM or only masks impaired pancreatic beta-cell function. Diabetes is diagnosed by a fasting plasma glucose of 7.8 mmol/L or more, or a two-hour post-load blood glucose of 10 mmol/L or more (Falkenberg & Finnstrom, 1994, p. 846). The latter value is approximately the renal threshold for glucose. Despite differences in the prevalence of NIDDM between populations, the summit of the inverted U curve consistently coincides with the renal threshold for glucose. One interpretation is that decompensation of beta-cell function occurs after the point where hyperglycaemia no longer signals the presence of insulin resistance to beta-cells. On the other hand, there may be 75 percent loss of beta-cell secretory capacity by the time the top of the curve is reached. Thus, the increased beta-cell demand induced by insulin resistance is associated with progressive loss of beta-cell function. Weight loss and insulin therapy in patients with NIDDM increase endogenous insulin secretion, although weight loss itself would not be expected to influence insulin secretion, and exogenous insulin, if anything, acutely inhibits endogenous insulin secretion (Falkenberg & Finnstrom, 1994, p. 846). Improved
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Some common words found in the essay are:
Falkenberg Finnstrom, Type II, Assessment Genetic, Wierenga Hewitt, NIDDM Insulin, Plummer Albert, Introduction Diabetes, type ii, Nursing Diagnosis, insulin resistance, Diagnosis Genetic, VI Conclusion, insulin secretion, diabetes mellitus, diabetic patients, ii diabetic patients, beta-cell function, ii diabetic, finnstrom 1994, nurse practitioner, falkenberg finnstrom, falkenberg finnstrom 1994, type ii diabetic, type ii diabetes, genetic environmental factors,
Approximate Word count = 2535
Approximate Pages = 10 (250 words per page)

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