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Cognitive Model of Antisocial Personality Disorder

ises from defective avoidance learning and/or defective autonomic nervous system structures and processes. It is concluded that, therefore, it is possible that the anomaly producing the disorder could be cognitive, involving not the autonomic but the central nervous system.

In relation to the foregoing conclusion, Gorenstein (1991) postulates those central nervous system structures and processes that could be involved in the etiology of antisocial personality. Specifically, Gorenstein cites involvement of the septal-hippocampal frontal system.

The assertion of the involvement of the septal-hippocampal frontal system in antipersonality disorder is postulated based on animal research showing that lesions to various structures in the system produce certain similarities in behavioral responses. The similarities between the lesioned animals and individuals with antisocial personality disorder are said to include:

1. Attenuated avoidance of incidental punishment.

2. Attenuated anticipation of aversive events.

3. Attenuated mediation of temporal intervals.

Neurological studies of individuals with antisocial personality disorder are then explored and discussed in an effort to show that psychopathic subjects (in both clinical and prison populations) showed performance deficits that could be attributed to defective functioning of the septal-hippocampal frontal system. The cited studies are then used to articulate the basic thesis of the cognitive model.

This basic thesis is that antisocial personality disorder is associated with a weakness in cognitive mediation, leaving the individual overly depende

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Cognitive Model of Antisocial Personality Disorder. (1969, December 31). In LotsofEssays.com. Retrieved 05:49, March 29, 2024, from https://www.lotsofessays.com/viewpaper/1689830.html